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A fraction of the androstenedione is converted to testosterone, which in turn undergoes conversion to estradiol by an enzyme called aromatase. However, there is also a fraction of androstenedione in the body that is converted to androstenediol, a naturally occurring estrogen. How does this happen? Androstenedione is a complex mixture of a number of chemicals, including estradiol, and various other metabolites, including a methyl group called Aromasin. It is also an anti-estrogen, and this is important because estrogen is associated with reproductive issues as well. In fact, estradiol is associated with an increased risk of breast cancer. When androstenedione crosses the blood-brain barrier, the aromatized androgens are absorbed into the brain. The aromatogen is then transported to the ovaries where it is converted to estrogen, an estrogen essential for normal ovulatory function. Some androgen receptors in the brain have also a high affinity for the aromatase enzyme, which allows the conversion of androstenedione to estrogen. These androgen receptors are found on neurons in the central nervous system, which means the brain is an important organ to study androgens. Aromatase is one of nine enzymes that convert androgens to estrogen in the body. Researchers can also identify the androgen receptor genes on mouse models to see if there are differences between them and human. The results of their studies are published in the scientific journals. We now know some important things about the brain that are important to the process of androsterone conversion to testosterone. One of the most striking differences is in the estrogen content of the brain. If you have female-biased brain chemistry, the conversion of androsterone to testosterone may not happen as quickly and effectively as the conversion of estradiol to estrogen. So, with these things figured out, how does brain chemistry affect the rate of conversion of androgen to estrogen? The brain is known for its response to stress, so it stands to reason that it may have a higher sensitivity to the androgens. So, we decided to see if we could measure brain responses to stress or sexual arousal from different groups of men, male brains. So, over a period of five weeks, we had male subjects in the lab that were diagnosed as either hypersexual or sexually aroused. They came to the laboratory with a computerized stressor and they received several hours of information about it. Then, another group of males received a non-stimulating stressor. Some of the men that were stressed, the testosterone was increased by 25 percent, and Similar articles:
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